Search results for " III"

showing 10 items of 562 documents

Numerical Procedures and their Practical Application in PV Module Analyses. Part IV: Atmospheric Transparency Parameters - Application

2020

Abstract The presented article relates to aspects of PV module testing using natural sunlight in outdoor conditions. It is a continuation of the article Part III: parameters of atmospheric transparency - determining and correlations. This article discusses the practical application of the indexes: atmosphere purity - k Tm , diffused component content - k s/o , beam clear sky index - K b - in testing various modules in outdoor conditions. Their influence on the conversion of modules made from various absorbers and various technologies is demonstrated. Their practical application in module testing in outdoor conditions is described and it - has been demonstrated that the results of the analys…

010302 applied physicsChemistrybusiness.industry020209 energysolar energyAtmospheric transparencysolar radiation spectrum02 engineering and technologysky clearness indexdiffused component content index01 natural sciencesEnergy engineeringPart iiiphotovoltaicsComponent (UML)0103 physical sciences0202 electrical engineering electronic engineering information engineeringGeneral Materials ScienceProcess engineeringbusinessEcological Chemistry and Engineering S
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Genome-Wide Profiling and Phylogenetic Analysis of the SWEET Sugar Transporter Gene Family in Walnut and Their Lack of Responsiveness to Xanthomonas …

2020

Following photosynthesis, sucrose is translocated to sink organs, where it provides the primary source of carbon and energy to sustain plant growth and development. Sugar transporters from the SWEET (sugar will eventually be exported transporter) family are rate-limiting factors that mediate sucrose transport across concentration gradients, sustain yields, and participate in reproductive development, plant senescence, stress responses, as well as support plant&ndash

0106 biological sciences0301 basic medicinephylogeny01 natural scienceslcsh:Chemistrywalnut blightTAL effectorType III Secretion Systems2.1 Biological and endogenous factorsAetiologylcsh:QH301-705.5SpectroscopyPlant Proteins<i>Xanthomonas</i>GeneticsGenomebiologyfood and beveragesSWEET sugar transportersGeneral MedicineSucrose transportComputer Science ApplicationsInfectious DiseasesMultigene Familygene familyJuglansXanthomonasPlant DevelopmentJuglansCatalysisInorganic Chemistry03 medical and health sciencesTAL effectorXanthomonasGeneticsGene familySugar transporterPhysical and Theoretical ChemistryMolecular BiologyGenePlant DiseasesChemical PhysicsOrganic ChemistryfungiMembrane Transport ProteinsBiological TransportXanthomonas arboricolaPlantbiology.organism_classification030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Gene Expression Regulationgene expressionOther Biological SciencesOther Chemical Sciences010606 plant biology & botanyInternational Journal of Molecular Sciences
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Arabidopsis RCD1 coordinates chloroplast and mitochondrial functions through interaction with ANAC transcription factors

2019

Reactive oxygen species (ROS)-dependent signaling pathways from chloroplasts and mitochondria merge at the nuclear protein RADICAL-INDUCED CELL DEATH1 (RCD1). RCD1 interacts in vivo and suppresses the activity of the transcription factors ANAC013 and ANAC017, which mediate a ROS-related retrograde signal originating from mitochondrial complex III. Inactivation of RCD1 leads to increased expression of mitochondrial dysfunction stimulon (MDS) genes regulated by ANAC013 and ANAC017. Accumulating MDS gene products, including alternative oxidases (AOXs), affect redox status of the chloroplasts, leading to changes in chloroplast ROS processing and increased protection of photosynthetic apparatus.…

0106 biological sciences0301 basic medicineretrograde signalingChloroplastsArabidopsisPlant BiologyMitochondrion01 natural sciencesElectron Transport Complex IIIGene Expression Regulation PlantArabidopsisOXIDATIVE STRESS-RESPONSETranscriptional regulationCYCLIC ELECTRON FLOWBiology (General)Nuclear proteinANAC transcription factors1183 Plant biology microbiology virologyreactive oxygen speciesbiologyChemistryRETROGRADE REGULATIONGeneral NeuroscienceQRNuclear Proteinsfood and beveragesGeneral MedicinePlants Genetically Modified:Science::Biological sciences [DRNTU]Cell biologyMitochondriaChloroplastviherhiukkasetMedicineSignal transductionmitochondrial functionsResearch ArticleSignal TransductionQH301-705.5SciencemitokondriotGenetics and Molecular BiologyGeneral Biochemistry Genetics and Molecular BiologyPROTEIN COMPLEXESSIGNALING PATHWAYS03 medical and health scienceschloroplastStress PhysiologicalALTERNATIVE OXIDASESkasvitENZYME-ACTIVITIESredox signalingTranscription factorarabidopsis RCD1General Immunology and MicrobiologybiokemiaArabidopsis Proteinsta1182Biology and Life Sciencesbiology.organism_classification030104 developmental biologyCELL-DEATHPLANT-MITOCHONDRIAA. thalianaGeneral BiochemistryRetrograde signalingGENES-ENCODING MITOCHONDRIALproteiinit010606 plant biology & botanyTranscription Factors
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Standardization proposal for the mapping of Caulerpa taxifolia expansion in the Mediterranean sea

1999

Fourteen years after the first observation of Caulerpa taxifolia (Valh) C. Agardh in the northwestern Mediterranean Sea, this green alga of tropical origin is now present in five countries (Spain, France, Monaco, Italy and Croatia). By the end of 1997, more than 46 km(2), at depths of between 0 and 50 m, were found to be affected by this expansion. A standardization of the cartographic procedure is proposed here. Such a standardization is necessary to compare maps produced by various organizations from different countries. For a given independent station, it is based on the definition of three levels of colonization:Level I refers to a station where one or several colonies less than 100 m a…

0106 biological sciencesCartographyCaulerpa taxifoliaMediterranean SeacartographieAquatic ScienceOceanography010603 evolutionary biology01 natural sciencesInvasive speciesSurface areaMediterranean seacartography; Caulerpa taxifolia; invasive species; Mediterranean seaMediterranean SeaColonization14. Life underwaterbiologyInvasive speciesEcology010604 marine biology & hydrobiologyUlvophyceaebiology.organism_classificationStandardizationGeographyMer MéditerranéePhysical geographyLevel iiLevel iiiStandardisationCaulerpa taxifoliaEspèces envahissantes
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Downregulation of thioredoxin-1-dependent CD95 S-nitrosation by Sorafenib reduces liver cancer

2020

Hepatocellular carcinoma (HCC) represents 80% of the primary hepatic neoplasms. It is the sixth most frequent neoplasm, the fourth cause of cancer-related death, and 7% of registered malignancies. Sorafenib is the first line molecular targeted therapy for patients in advanced stage of HCC. The present study shows that Sorafenib exerts free radical scavenging properties associated with the downregulation of nuclear factor E2-related factor 2 (Nrf2)-regulated thioredoxin 1 (Trx1) expression in liver cancer cells. The experimental downregulation and/or overexpression strategies showed that Trx1 induced activation of nitric oxide synthase (NOS) type 3 (NOS3) and S-nitrosation (SNO) of CD95 rece…

0301 basic medicine:Anatomy::Cells::Cells Cultured::Cell Line::Cell Line Tumor [Medical Subject Headings]Factor 2 relacionado con NF-E2Regulación hacia abajomedicine.medical_treatment[SDV]Life Sciences [q-bio]Clinical Biochemistry:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Thioredoxins [Medical Subject Headings]ApoptosisBiochemistry:Phenomena and Processes::Chemical Phenomena::Biochemical Phenomena::Biochemical Processes::Nitrosation [Medical Subject Headings]Targeted therapyNeoplasias hepáticas:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]Mice0302 clinical medicineThioredoxins:Organisms::Eukaryota::Animals [Medical Subject Headings]lcsh:QH301-705.5Cell proliferationlcsh:R5-920GSNORChemistry:Diseases::Neoplasms::Neoplasms by Site::Digestive System Neoplasms::Liver Neoplasms [Medical Subject Headings]Liver NeoplasmsSorafenibFas receptor3. Good healthHepatocellular carcinomaCD95Liver cancerlcsh:Medicine (General)NOS3Liver cancerCarcinoma hepatocelularResearch Papermedicine.drugSorafenibHepatocarcinomaProliferación celularCarcinoma HepatocellularNitrosationDown-RegulationMice Nude[SDV.CAN]Life Sciences [q-bio]/CancerAntineoplastic AgentsNrf203 medical and health sciencesDownregulation and upregulationCell Line TumormedicineAnimalsHumansS-NitrosoglutatiónTiorredoxinas:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Growth Processes::Cell Proliferation [Medical Subject Headings]:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Rodentia::Muridae::Murinae::Mice [Medical Subject Headings]:Diseases::Neoplasms::Neoplasms by Site::Digestive System Neoplasms::Liver Neoplasms::Carcinoma Hepatocellular [Medical Subject Headings]:Phenomena and Processes::Chemical Phenomena::Biochemical Phenomena::Biochemical Processes::Down-Regulation [Medical Subject Headings]Cell growthPhenylurea CompoundsOrganic Chemistry:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Therapeutic Uses::Antineoplastic Agents [Medical Subject Headings][SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Rodentia::Muridae::Murinae::Mice::Mice Mutant Strains::Mice Nude [Medical Subject Headings]medicine.diseasedigestive system diseases030104 developmental biologylcsh:Biology (General)ApoptosisDownregulation:Chemicals and Drugs::Organic Chemicals::Hydrocarbons::Hydrocarbons Cyclic::Hydrocarbons Aromatic::Benzene Derivatives::Phenylurea Compounds [Medical Subject Headings][SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyCancer researchÓxido nítrico sintasa de tipo III030217 neurology & neurosurgery
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New Therapeutic Implications of Endothelial Nitric Oxide Synthase (eNOS) Function/Dysfunction in Cardiovascular Disease

2019

The Global Burden of Disease Study identified cardiovascular risk factors as leading causes of global deaths and life years lost. Endothelial dysfunction represents a pathomechanism that is associated with most of these risk factors and stressors, and represents an early (subclinical) marker/predictor of atherosclerosis. Oxidative stress is a trigger of endothelial dysfunction and it is a hall-mark of cardiovascular diseases and of the risk factors/stressors that are responsible for their initiation. Endothelial function is largely based on endothelial nitric oxide synthase (eNOS) function and activity. Likewise, oxidative stress can lead to the loss of eNOS activity or even &ldquo;uncoupli…

0301 basic medicineAdipose tissueReview030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeendothelial dysfunctionEpigenesis Geneticlcsh:Chemistry0302 clinical medicineEnoscardiovascular diseaseeNOS uncouplingoxidative stressEndothelial dysfunctionlcsh:QH301-705.5Spectroscopyenvironmental stressorsbiologyGeneral MedicineComputer Science Applicationsmedicine.anatomical_structureCardiovascular Diseasesmedicine.symptomOxidation-ReductionCell signalingEndotheliumNitric Oxide Synthase Type IIIInflammationModels BiologicalCatalysisInorganic Chemistry03 medical and health scienceslife style/behavioral health risk factorsmedicineAnimalsHumansPhysical and Theoretical ChemistryMolecular Biologybusiness.industryOrganic Chemistrymedicine.diseasebiology.organism_classification030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Socioeconomic FactorsinflammationSoluble guanylyl cyclasebusinessOxidative stressInternational Journal of Molecular Sciences
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Treatment for hereditary angioedema with normal C1-INH and specific mutations in the F12 gene (HAE-FXII).

2016

Hereditary angioedema with normal C1 esterase inhibitor and mutations in the F12 gene (HAE-FXII) is associated with skin swellings, abdominal pain attacks, and the risk of asphyxiation due to upper airway obstruction. It occurs nearly exclusively in women. We report our experience treating HAE-FXII with discontinuation of potential trigger factors and drug therapies. The study included 72 patients with HAE-FXII. Potential triggers included estrogen-containing oral contraceptives (eOC), hormonal replacement therapy, or angiotensin-converting enzyme inhibitors. Drug treatment comprised plasma-derived C1 inhibitor (pdC1-INH) for acute swelling attacks and progestins, tranexamic acid, and danaz…

0301 basic medicineAdultMalemedicine.medical_specialtyExacerbationAdolescentImmunologyAngiotensin-Converting Enzyme InhibitorsGastroenterologyChemopreventionC1-inhibitorHereditary Angioedema Type III03 medical and health sciencesYoung Adult0302 clinical medicineRisk FactorsInternal medicinemedicineImmunology and AllergyHumansHereditary Angioedema Type IIIChildAgedDanazolbiologybusiness.industryEstrogensMiddle Agedmedicine.diseaseSurgeryDiscontinuation030104 developmental biologyTreatment Outcome030228 respiratory systemQuinaprilHereditary angioedemaFactor XIIMutationbiology.proteinDisease ProgressionFemalebusinessComplement C1 Inhibitor ProteinTranexamic acidBiomarkersmedicine.drugAllergy
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Analysis of cold activation of the contact system in hereditary angioedema with normal C1 inhibitor.

2021

Hereditary angioedema (HAE) attacks are caused by excessive activation of the contact system. Understanding how the contact system is activated in HAE, especially in patients with normal C1 inhibitor (HAEnCI), is essential to effectively treat this disease. Contact system activation involves the cleavage of several proteins including Factor XII (FXII), high molecular weight kininogen (HK), prekallikrein, sgp120 (ITIH4) and C1 inhibitor (C1-INH) before the subsequent generation of bradykinin that mediates HAE. In this study, we evaluated the fragmentation and enzymatic activity of contact system proteins in HAEnCI plasma samples before and after contact system activation induced by incubatio…

0301 basic medicineAdultMalemedicine.medical_specialtyHigh-molecular-weight kininogenImmunologyProteinase Inhibitory Proteins SecretoryBradykininBradykininC1-inhibitorHereditary Angioedema Type III03 medical and health scienceschemistry.chemical_compoundYoung Adult0302 clinical medicineInternal medicinemedicineHumansFragmentation (cell biology)Molecular BiologyBlood CoagulationFactor XIIbiologyKininogensPrekallikreinPrekallikreinEstrogensPlasminogenKallikreinMiddle Agedmedicine.diseaseCold Temperature030104 developmental biologyEndocrinologychemistryHereditary angioedemaFactor XIIbiology.proteinFemaleKallikreinsComplement C1 Inhibitor Protein030215 immunologyMolecular immunology
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New 1,4-Dihydropyridines Down-regulate Nitric Oxide in Animals with Streptozotocin-induced Diabetes Mellitus and Protect Deoxyribonucleic Acid agains…

2015

Diabetes mellitus (DM) and its complications cause numerous health and social problems throughout the world. Pathogenic actions of nitric oxide (NO) are responsible to a large extent for development of complications of DM. Search for compounds regulating NO production in patients with DM is thus important for the development of pharmacological drugs. Dihydropyridines (1,4-DHPs) are prospective compounds from this point of view. The goals of this study were to study the in vivo effects of new DHPs on NO and reactive nitrogen and oxygen species production in a streptozotocin (STZ)-induced model of DM in rats and to study their ability to protect DNA against nocive action of peroxynitrite. STZ…

0301 basic medicineBlood GlucoseMaleDihydropyridinesNitric Oxide Synthase Type IIIXanthine DehydrogenaseDown-RegulationNitric Oxide Synthase Type IIDHPS030204 cardiovascular system & hematologyPharmacologyToxicologyEndothelial NOSKidneyNitric OxideProtective AgentsNitric oxideDiabetes Mellitus Experimental03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePeroxynitrous AcidmedicineAnimalsRats WistarReactive nitrogen speciesPharmacologybiologyGeneral MedicineDNAStreptozotocinReactive Nitrogen SpeciesRatsNitric oxide synthasePeroxynitrous acid030104 developmental biologyBiochemistrychemistryLiverbiology.proteinReactive Oxygen SpeciesPeroxynitritemedicine.drugBasicclinical pharmacologytoxicology
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Haploinsufficiency of the Primary Familial Brain Calcification Gene SLC20A2 Mediated by Disruption of a Regulatory Element

2020

OBJECTIVE Primary familial brain calcification (PFBC) is a rare cerebral microvascular calcifying disorder with diverse neuropsychiatric expression. Five genes were reported as PFBC causative when carrying pathogenic variants. Haploinsufficiency of SLC20A2, which encodes an inorganic phosphate importer, is a major cause of autosomal-dominant PFBC. However, PFBC remains genetically unexplained in a proportion of patients, suggesting the existence of additional genes or cryptic mutations. We analyzed exome sequencing data of 71 unrelated, genetically unexplained PFBC patients with the aim to detect copy number variations that may disrupt the expression of core PFBC-causing genes. METHODS Afte…

0301 basic medicineBrain DiseasesDNA Copy Number VariationsSodium-Phosphate Cotransporter Proteins Type IIIHEK 293 cellsBrainHaploinsufficiencyBiologyMolecular biologyReverse transcriptase03 medical and health sciencesHEK293 Cells030104 developmental biology0302 clinical medicineNeurologyMutationHumansNeurology (clinical)Copy-number variationAlleleHaploinsufficiencyEnhancerGene030217 neurology & neurosurgeryExome sequencingMovement Disorders
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